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Articles by Zhiqiang LI
Total Records ( 2 ) for Zhiqiang LI
  Yucheng WU , Gang RONG , Jixiong LI , Luheng ZHANG and Zhiqiang LI
  In petrochemical plants, schedulers have to determine how much newly received raw material should be piped into reception tanks which stored previously received raw material and mix raw material with desirable composition proportion to produce products with due dates. To address the challenge, we established an optimization model to deal with short-term production scheduling integrated with raw materials mixing process and introduced a strategy to make due dates of products demands flexible that can be adjusted at minimum costs while original ones cannot be met. A novel bounding algorithm is developed to determine hard bounds of variables in bilinear terms of the model, which makes the proposed mixed integer nonlinear programing (MNILP) model can be solved effectively by using the global solution approach based on piecewise defined convex envelops. Finally, a study case of petrochemical production is presented to illustrate the approaches proposed in the paper.
  Fei Guo , Yuanzhi Wang , Chuangfu Chen , Hui Zhang , Jun Qiao , Yan Ren , Junbo Zhang and Zhiqiang Li
  TFSS is an important virulence factor of Brucella, organized as one operon containing 12 different across the cell wall bacterial proteins among which VirB5 regulates the host phagocytosis of Brucella and the transportation of Brucella in the host cells. This study has constructed cDNA library from Brucella melitensis 16M-infected murine macrophage Raw264.7, identified and confirmed the interaction between Brucella VirB5 and FTH1 of RAW264.7 using yeast two-hybrid and Co-Immunoprecipitation (Co-IP) technologies. Subsequently, the morphological changes and the expression of apoptosis-related genes in Brucella-infected RAW264.7 cells have been investigated with Electron Microscope (EM) and real-time quantitative RT-PCR, respectively. The present study has demonstrated that VirB5 and FTH1 play important roles in intracellular parasitism of Brucella and inhibition of FHT1 expression accelerates the apoptosis of macrophage.
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