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Articles by Min Liu
Total Records ( 8 ) for Min Liu
  Min Liu , J. B. Sartain , L. E. Trenholm and G. L. Miller
  Phosphorus fertilization of St. Augustinegrass [Stenotaphrum secondatum (Walt.) Kuntze] is performed on the basis of soil tests designed for agronomic crops. The objectives of this study were to determine the critical minimum P application rate and soil and tissue P concentration of St. Augustinegrass grown in sandy soils. Three studies were conducted in a glasshouse using a Pomona sand, two Tavares sands, and a Pottsburg sand. Established pots of ‘Floratam’ St. Augustinegrass received P at 0, 0.14, 0.27, 0.54, and 1.09 g m–2 every four weeks for 16 weeks for Study 1 and 12 weeks for Studies 2 and 3. Critical minimum tissue P concentration was determined to be 1.8 g kg–1 on dry weight basis. Minimum P application rate was 0.14 g m–2 4-wk–1 to obtain acceptable turfgrass quality and growth rate. Additional growth was not observed when Mehlich-1 P level was greater than 7, 8, 9, and 5 mg kg–1 for Pomona sand, Tavares sand (a), Pottsburg sand, and Tavares sand (b), respectively. Phosphorus fertilization would not be recommended if soil Mehlich-1 P concentration is above 10 mg kg–1 for sandy soils or tissue P level is beyond 1.8 g kg–1 on dry weight basis. Phosphorus application of 0.14 g m–2 4-wk–1 is recommended when P is required.
  Min Liu and Ying Li
  Due to its limited resources and other objective factors and subjective attitude of the impact of peer nodes in the network often exhibit selfishness. Therefore, encourage selfish nodes cooperate to detect and become an important research by allowing nodes to freely express their subjective forward attitude. To achieve the detection of selfish nodes, it is taking into account not only the quality of the link and node energy and other objective factors that determine the path forward probability and taking into account the path under the influence of selfish nodes forwarding the subjective probability. To select the path with the highest probability of integrated transponder, it is reducing the impact of selfish nodes when the node selfishness spend time re-designed a punishment mechanism based load balancing to encourage cooperation model participation and cooperation of nodes according to the degree of harm node selfishness. The right is taken appropriate punitive measures between nodes monitoring mechanism and strict punishment mechanism to ensure the implementation of strategic defense. The simulation results show that the detection and load balancing not only in the energy constrained and rational selfish nodes case seeking to the appropriate route, but also to inspire too selfish nodes actively participate in the network.
  Ming Qian , Min Liu , Mengna Duan , Haiyang Zhang , Zhe Wu and Yanmin Zhou
  Substance P (SP) is known to play an important role in bone mineralization. However, little is known about the potential of substance P as an osteogenic inducer in human Adipose-Derived Stem Cells (hADSCs) in vitro. Therefore, the aim of this study to investigate the effects of substance P on proliferation and osteogenic differentiation of hADSCs in vitro. Human Adipose-Derived Stem Cells (hADSCs) derived from wasted adipose tissue in liposuction operation were cultured. Expression of the SP receptor (NK1) was detected by RT-PCR. Effects of SP on proliferation and differentiation of hADSCs were studied, moreover alkaline phosphatase activity was also assayed. It was found that the NK1 receptor was expressed in hADSCs. SP stimulated the proliferation of hADSCs in a concentration-dependent manner. Higher concentrations (10G8 M) of SP increased alkaline phosphatase activity and enhanced differentiated in hADSCs. NK1 receptors are expressed by hADSCs and SP stimulates osteoblast differentiation and function in vitro. These findings strengthen the knowledge about the role of SP in hADSCs.
  Min Liu , D. E. Kissel , L. S. Sonon , M. L. Cabrera and P. F. Vendrell
  Incubation of soil with CaCO3 is generally considered a reliable method to determine the lime requirement (LR) of acid soils. Because of their considered reliability, these incubations are often used to calibrate buffer methods; however, one study reported that the use of room temperature incubation with CaCO3 overestimated the actual LR determined by field testing. The objective of this study was to compare the pH change following CaCO3 incubations for 60 d with those following 3-d incubations with Ca(OH)2 and to determine the possible role of soil N reactions causing any differences in pH change. Seventeen soils were incubated with either CaCO3 for 60 d at approximately 85% field capacity or for 3 d with an equivalent amount of Ca(OH)2 solution plus water to maintain a 1:1 soil/solution ratio. Both were incubated at room temperature (23 ± 2°C), followed by measurement of pH (1:1 in water). Ammonium-N and NO3–N were analyzed at Days 0 and 60 of the incubation. Soil pH was lower following the 60-d CaCO3 incubation than after the 3-d incubation with Ca(OH)2. The analysis of N transformations indicated that positive values of H+ (more H+ was produced than consumed) were generated from nitrification after 60 d of incubation in 14 out of 17 soils. Furthermore, incubations with soils that have been air dried produced a flush of nitrification that increased the ionic strength and decreased pH even further. These effects from long-term incubation would erroneously increase the LR. Incubation with Ca(OH)2 for 2 to 4 d avoids these errors.
  Wenpu Zhao , Min Liu and Keith L. Kirkwood
  AU-rich elements (AREs) in the 3`-untranslated region (UTR) of unstable mRNA dictate their degradation or mediate translational repression. Cell signaling through p38α MAPK is necessary for post-transcriptional regulation of many pro-inflammatory cytokines. Here, the cis-acting elements of interleukin-6 (IL-6) 3`-UTR mRNA that required p38α signaling for mRNA stability and translation were identified. Using mouse embryonic fibroblasts (MEFs) derived from p38α+/+ and p38α–/– mice, we observed that p38α is obligatory for the IL-1-induced IL-6 biosynthesis. IL-6 mRNA stability is promoted by p38α via 3`-UTR. To understand the mechanism of cis-elements regulated by p38α at post-transcriptional level, truncation of 3`-UTR and the full-length 3`-UTR with individual AUUUA motif mutation placed in gene reporter system was employed. Mutation-based screen performed in p38α+/+ and p38α–/– mouse embryonic fibroblast cells revealed that ARE1, ARE2, and ARE5 in IL-6 3`-UTR were targeted by p38α, and truncation-based screen showed that IL-6 3`-UTR-(56–173) was targeted by p38α to stable mRNA. RNA secondary structure analysis indicated that modulated reporter gene expression was consistent with predicted secondary structure changes.
  Dongsheng Wu , Wenlong Huang , Peter M. Richardson , John V. Priestley and Min Liu
  Canonical transient receptor potential (TRPC) receptors are Ca2+-permeable cation channels that have a variety of physiological functions and may be involved in neuronal development and plasticity. We investigated the expression profile of TRPC channels in adult rat dorsal root ganglia (DRG) after nerve injury and examined the role of TRPC4 in neurite outgrowth in cultured DRG neurons. Sciatic nerve transection and microinjection of dibutyryl cAMP were employed to induce axonal regeneration in vivo. TRPC4 mRNA was significantly increased whereas TRPC1, TRPC3, TRPC6, and TRPC7 remained unaltered after nerve injury or dibutyryl-cAMP microinjection. The increases in TRPC4 transcript and protein were transient with maximal levels reached at 2 or 7 days, respectively. In addition, TRPC4 transcript in ND7/23 and NDC cells, hybrid cell lines derived from neonatal DRG and neuroblastoma, was substantially increased on differentiation, characterized by neurite outgrowth. In adult DRG, TRPC4 immunoreactivity was found in small and large neurons, and nerve injury increased the number of TRPC4-immunoreactive cells, particularly in large neurons. TRPC4 immunoreactivity was present in growth cones at various stages of DRG neurite outgrowth in vitro. Suppression of TRPC4 by a specific small interfering RNA or antisense significantly reduced the length of neurites in cultured DRG neurons. Expression of short hairpin RNA significantly down-regulated TRPC4 protein level and shortened neurite lengths in differentiated ND7/23 cells. The reduction in neurite lengths in ND7/23 cells was rescued by overexpression of human TRPC4. Our results suggest that TRPC4 contributes to axonal regeneration after nerve injury.
  Jinmin Gao , Lihong Huo , Xiaoou Sun , Min Liu , Dengwen Li , Jin- Tang Dong and Jun Zhou
  The familial cylindromatosis tumor suppressor CYLD is known to contain three cytoskeleton-associated protein glycine-rich (CAP-Gly) domains, which exist in a number of microtubule-binding proteins and are responsible for their association with microtubules. However, it remains elusive whether CYLD interacts with microtubules and, if so, whether the interaction is mediated by the CAP-Gly domains. In this study, our data demonstrate that CYLD associates with microtubules both in cells and in vitro, and the first CAP-Gly domain of CYLD is mainly responsible for the interaction. Knockdown of cellular CYLD expression dramatically delays microtubule regrowth after nocodazole washout, indicating an activity for CYLD in promoting microtubule assembly. Our data further demonstrate that CYLD enhances tubulin polymerization into microtubules by lowering the critical concentration for microtubule assembly. In addition, we have identified by wound healing assay a critical role for CYLD in mediating cell migration and found that its first CAP-Gly domain is required for this activity. Thus CYLD joins a growing list of CAP-Gly domain-containing proteins that regulate microtubule dynamics and function.
  Min Liu , Ritu Aneja , Xiaodong Sun , Songbo Xie , Hongxia Wang , Xiaojing Wu , Jin-Tang Dong , Minggang Li , Harish C. Joshi and Jun Zhou
  Eg5 is a motor protein of the kinesin family that is critical for spindle assembly during mitosis and has recently been implicated in tumorigenesis. It is largely unknown how Eg5 expression is regulated in cells. In this study, we present the first evidence that the cellular Eg5 level is down-regulated by Parkin, an E3 ubiquitin ligase well known for its role in the development of Parkinson disease. Our data show that Parkin does not trigger Eg5 protein degradation through the ubiquitin-proteasome pathway. Instead, Parkin represses Eg5 gene transcription by blocking c-Jun binding to the activator protein 1 site present in the Eg5 promoter. Our data further show that Parkin inactivates c-Jun NH2-terminal kinase (JNK), resulting in decreased phosphorylation of c-Jun. The inactivation of JNK is further mediated by multiple monoubiquitination of Hsp70. Importantly, both the ubiquitination of Hsp70 and the subsequent inactivation of the JNK-c-Jun pathway are crucial for Parkin to down-regulate Eg5 expression. These results thus uncover a novel function for Parkin in modulating the expression of Eg5 through the Hsp70-JNK-c-Jun signaling pathway.
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