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Articles by Lin Dong
Total Records ( 2 ) for Lin Dong
  Lianjun Liu , Bin Liu , Lihui Dong , Jie Zhu , Haiqin Wan , Keqin Sun , Bin Zhao , Haiyang Zhu , Lin Dong and Yi Chen
  In situ FT-IR was employed to investigate CO or/and NO interaction with CuO supported on Ce0.67Zr0.33O2 (hereafter denoted as CZ) catalysts. The physicochemical properties of CuO–CZ were also studied by combination of XRD, TPR and NO + CO activity tests. The results indicated that the dispersed CuO species were the main active components for this reaction. The catalysts showed different activities and selectivities at low and high temperatures, which should be resulted from the reduction of dispersed copper oxide species. This reaction went through different mechanisms at low and high temperatures due to the change of active species. FT-IR results suggested: (1) CO was activated by oxygen originating from CZ support, which led to surface carbonates formation, and partial dispersed CuO was reduced to Cu+ species above 150 °C; (2) NO interacted with the dispersed CuO and formed several types of nitrite/nitrate species, whereas crystalline CuO made little contribution to the formation of new NO adsorbates; (3) NO was preferentially adsorbed on CuO–CZ catalysts compared with CO in the reactants mixture. These adsorbed nitrite/nitrate species exhibited different thermal stability and reacted with CO at 250 °C. As a result, a possible mechanism was tentatively proposed to approach NO reduction by CO over CuO–CZ catalyst.
  Wenjing Zhang , Haiting An , Feilong Zhang , Lin Dong , Qi Wang , Ruijun Su , Yanjing Qian and Xiaoli Gong
  Parkinson’s Disease (PD) is a neurodegenerative disorder characterized by the progressive loss of dopaminergic (DA) neurons in the Substantia Nigra pars Compacta (SNc). There is no satisfactory therapeutic strategy for PD as yet. Here, triptolide (T10), a major active compound extracted from Tripterygium wilfordii Hook. f. (TWHF), a Traditional Chinese Medicine (TCM) used to treat autoimmune diseases, was found to significantly diminish the number of abnormal rotations induced by apomorphine in 6-hydroxydopamine (6-OHDA)-lesioned PD rat model and protected DA neurons in SNc from 6-OHDA toxicity. Triptolide treatment was found to significantly suppress the activation of microglia and reduce the levels of oxidation products of proteins and lipids in the striatonigral systems of the PD model rats. Results suggest that triptolide might be a suitable lead compound in drug discovery for PD therapy.
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