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Articles by Ya Liu
Total Records ( 3 ) for Ya Liu
  Meiling Zhang , Tao Gui , Yunsheng Li , Chao Wei , Nian Zhan , Zhen Chen , Yuanliang Zhang , Ya Liu , Qing Jia , Jianping Ding , Xiaorong Zhang and Yunhai Zhang
  This study was conducted to explore efficient and cost-effective protocols for isolation and culture of mammary epithelial cells from goats during gestation period (non-lactating). Tissues collected from the mammary glands of a Huanghuai white goat, at gestation day 60 were cultured in the presence of fetal bovine serum, insulin-transferrin-selenium and epidermal growth factor. Cell lines were purified using differential enzyme digestion, high density cultivation and continuous passaging. And subsequently characterized by immunofluorescence staining, cell growth curve, oil red O staining and karyotype analysis. In conclusion, a goat mammary epithelial cell line was successfully established and shown to exhibit normal conformation, growth characteristics and lactation function by using improved sample collection and in vitro culture methods. This study provides a new and efficient cell culture protocol, which may facilitate research into the development and function of goat mammary glands and the development of bioreactors.
  Gongliang Liu , Wenjing Kang , Bo Li and Ya Liu
  The unique characteristics of the underwater acoustic communication channel, such as three dimensional volumes of the environment and the limited energy, make it necessary to design and develop new routing algorithms. In this study, we propose an innovative energy-efficient multi-hop routing scheme for three-dimensional sensor networks based on compressed sensing. During each frame, a randomly chosen subset of nodes participates in the sensing process instead of all nodes delivering their message to the Sink node. Every chosen node finds a tour to the Sink node. As the message travels through the tour, each node computes the product of its sensing data and a random weighted coefficient and adds value to the intermediate result received from the last node. Analysis and simulation results show that our proposed algorithms are able to give an accurate approximation of the monitoring field and prolong network lifetime.
  Fengsong Wang , Peng Xia , Fang Wu , Dongmei Wang , Wei Wang , Tarsha Ward , Ya Liu , Felix Aikhionbare , Zhen Guo , Michael Powell , Bingya Liu , Feng Bi , Andrew Shaw , Zhenggang Zhu , Adel Elmoselhi , Daiming Fan , Timothy L. Cover , Xia Ding and Xuebiao Yao
  Helicobacter pylori persistently colonize the human stomach and have been linked to atrophic gastritis and gastric carcinoma. Although it is well known that H. pylori infection can result in hypochlorhydria, the molecular mechanisms underlying this phenomenon remain poorly understood. Here we show that VacA permeabilizes the apical membrane of gastric parietal cells and induces hypochlorhydria. The functional consequences of VacA infection on parietal cell physiology were studied using freshly isolated rabbit gastric glands and cultured parietal cells. Secretory activity of parietal cells was judged by an aminopyrine uptake assay and confocal microscopic examination. VacA permeabilization induces an influx of extracellular calcium, followed by activation of calpain and subsequent proteolysis of ezrin at Met469-Thr470, which results in the liberation of ezrin from the apical membrane of the parietal cells. VacA treatment inhibits acid secretion by preventing the recruitment of H,K-ATPase-containing tubulovesicles to the apical membrane of gastric parietal cells. Electron microscopic examination revealed that VacA treatment disrupts the radial arrangement of actin filaments in apical microvilli due to the loss of ezrin integrity in parietal cells. Significantly, expression of calpain-resistant ezrin restored the functional activity of parietal cells in the presence of VacA. Proteolysis of ezrin in VacA-infected parietal cells is a novel mechanism underlying H. pylori-induced inhibition of acid secretion. Our results indicate that VacA disrupts the apical membrane-cytoskeletal interactions in gastric parietal cells and thereby causes hypochlorhydria.
 
 
 
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