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Articles by Y. Ebrahim Nezhad
Total Records ( 3 ) for Y. Ebrahim Nezhad
  T. Vahdatpour , K. Nazer Adl , Y. Ebrahim Nezhad , N. Mahery Sis , S. R. Riyazi and S. Vahdatpour
  This study was conduced to determine effects of blood corticosterone (CS) increasing on some physiological parameters and performance of boiler chickens. To avoid treatment of birds with various forms of stress with administration of CS a model was developed to study of mimicked stress in chickens. Total 180 one-day old chicks of the cobb-500 strain from male sex were placed in 12 pens. CS at 4 levels (0, 10, 20 and 30 mg L-1) in drinking water was provided ad libitum between 1 to 49 days of age. Continuous intake of CS for 49 days caused increasing in serum glucose, cholesterol, triglycerides, high and low density lipoprotein and mortality. Final body weight, total feed intake and abdominal fat deposition were decreased, whereas feed conversion ratio was constant. The relative weights of major immunobiological organs including spleen, thymus and bursa of fabricius were decreased (p<0.05). Numerically, weights of selected visceral organs especially liver were elevation in all groups that received higher levels of CS. Therefore, it seems that CS intake is an alternative tool and useful test for assess the effects of physical, psychological and physiological stress in researches on broiler chickens.
  M. Fathi , K. Nazer adl , Y. Ebrahim Nezhad , H. Aghdam Shahryar , M. Daneshyar and T. Tanha
  The present study examined the possible role of reactive oxygen species in the pathogenesis of heart failure in broilers. The experiment was conducted with 160, 1 day old male broilers (Ross 308) to investigate to clarify the mechanism of cell injury causing the pathogenesis of PHS syndrome. The chickens were divided in 2 groups of 4 replicates and 20 chicks for any replicate. One group of these chickens was raised in Normal Temperature (NT) treatment and the other group was raised in Cold Temperature (CT) treatment for induce pulmonary hypertension syndrome. Mortality was inspected to determine cause of death and diagnose of heart failure. Hematological, biochemical and pathological tests were used to determine the incidence of PHS including total Red Blood Cell (RBC), Hemoglobin (HGB), Hematocrit (HCT), release of Alanine Transaminase (ALT), Aspartate Transaminase (AST), Lactate Dehydrogenase (LDH) and a best indicator of lipid oxidation subsequent to generated oxidative stress was Malondialdehyde (MDA). Sampling of blood and liver tissue were determined at day 21 and 42. At end of the experiment (week 6), 2 chicks from each replicate were randomly selected and slaughtered. The heart was removed; the right ventricle was dissected away from the left ventricle and septum then ratio of Right Ventricle weight to Total Ventricle weight (RV/TV) calculated too. The results of the experiment indicated that there was a significant difference in RBC, HBG at 21 and HGB, RBC, HCT at 42 between groups as CT group had a greater (p<0.05) HGB, RBC at 21 and HBG, RBC, HCT at 42. However, there was no significant difference in ALT, AST and LDH plasma levels between groups at day 21, CT group had greater (p<0.05) levels in AST, ALT and LDH at day 42. The levels malondialdehyde equivalents an indicator of lipid oxidation sub-sequent to generated oxidative stress at plasma and liver tissue was significantly higher (p<0.05) in CT group at day 21 and 42. RV/TV ratio and mortality due to ascites, also were significantly affected by treatments as CT group had greater (p<0.05) RV/TV and mortality due to ascites mortality percentage compared to NT group. In conclusion, the results indicated that the deteriration of heart function in modern fast growing broilers in the experimental model is associated with oxidative stress leading to lipid peroxidation and reactive oxygen species may be involved in the pathogenesis of the pulmonary hypertension syndrome in broilers chickens.
  M. Fathi , K. Nazeradl , Y. Ebrahim Nezhad , H. Aghdam Shahryar , M. Daneshyar and T. Tanha
  Major objectives of this study were to assess antioxidant protection in plasma and whole liver in broilers with pulmonary hypertension syndrome. The experiment was conducted with 160, 1 day old male broilers (Ross 308). The chickens were divided in 2 groups of 4 replicates and 20 chicks for any replicate. One group of these chickens was raised in normal temperature (control without PHS) treatment and the other group was raised in cold temperature (with PHS) treatment for induce pulmonary hypertension syndrome. Mortality was inspected to determine cause of death and diagnose of PHS. For evaluation of antioxidant status, the parameters that were determined in plasma and liver include: Glutathione Peroxidase (GPX), Superoxide Dismutase (SOD), Total Antioxidant Status (TAS) and Malondialdehyde (MDA). Sampling of blood and liver tissue were determined at day 21 and 42. At end of the experiment (week 6), 2 chicks from each replicate were randomly selected and slaughtered. The heart was removed, the right ventricle was dissected away from the left ventricle and septum then ratio of Right Ventricle weight to Total Ventricle weight (RV/TV) calculated too. The results of the experiment indicated that in plasma (GPX) and (SOD) activity were elevated in broilers with PHS compared to controls. It was also, PHS group had low TAS compared to control. But in liver, just GPX (not for SOD) was elevated in PHS group than control. The levels malondialdehyde equivalents an indicator of lipid oxidation subsequent to generated oxidative stress at plasma and liver tissue was significantly higher in PHS group compared control. RV/TV ratio and mortality due to ascites, also were significantly affected by treatments as PHS group had greater RV/TV and mortality due to ascites mortality percentage compared to control.
 
 
 
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