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Articles by S. Ghazanfari
Total Records ( 2 ) for S. Ghazanfari
  S. Ghazanfari , K. Nobari and T. Yamauchi
  Adiponectin is an adipokine hormone that influences several metabolic functions including glucose utilization, lipogenesis, energy homeostasis and immunity by signaling through two distinct receptors, AdipoR1 and AdipoR2. AdipoR1 is abundantly expressed in skeletal muscle, whereas AdipoR2 is predominantly expressed in the liver. AdipoR1 and AdipoR2 genes are ubiquitously expressed in chicken tissues and that their expression is altered by feed deprivation in the anterior pituitary gland and adipose tissue. Adiponectin and its receptors expressed in chicken testis. Suggesting that adiponectin either produced locally or from other sources can influence testicular function. These findings indicate that sexual maturation is associated with the up-regulation of adiponectin receptor gene expression in the chicken testis. Also, adiponectin, AdipoR1 and AdipoR2 mRNAs are expressed in chicken theca and granulose cells from preovulatory follicles. Adiponectin is highly expressed in theca cells as compared to granulose cells and could be involved in the development of chicken preovulatory follicle. Differences in the adiponectin expression level observed in different tissues suggest that the adiponectin gene has broad functions in the metabolism process. In order to reveal the function and mechanism of action of adiponectin in life, more considerable and penetrating work should be pursued.
  S. Ghazanfari , H. Kermanshahi , M.R. Nassiry , A. Golian , A.R.H. Moussavi and A. Salehi
  The aim of this study was to investigate the effects of feed restriction and different energy and protein contents of the diet on performance and growth hormone concentration in broiler chicken. Five hundred and seventy six day old Ross male broiler chicks were used in a 2x2x3 factorial arrangements in a completely randomized design experiment. Feeding programs consisted of ad libitum and Skip-a-Day (SAD) feed restriction, two energy levels (3100 and 2800 kcal ME kg-1) and three protein levels (22.3, 19.3 and 16.3% CP). Feed restriction (SAD) was applied during 22-32 d of age. Corn-soybean meal based diets containing vegetable oil were used. Body weight and feed intake were recorded weekly. At 21, 32 and 49 day of age, one bird from four replicate of each treatment was selected randomly to collect blood sample and then carcass, breast and thigh weight were measured. Blood samples assayed for Growth Hormone (GH) concentration by RIA. Feed restriction decreased feed intake and body weight gain (p<0.001) of birds during 22-32 and feed intake during 32- 49 day of age, while body weight gain was not affected during this period. Also, feed restriction decreased Feed Conversion Ratio (FCR) (p<0.01) and body weight gain (p<0.001) during 22-32 day of age. Feed intake (p<0.001) and body weight gain increased in broilers fed on low-energy diets compared with those fed on high-energy diets during different periods. Increasing levels of protein increased feed intake (p<0.001), body weight gain and improved FCR (p<0.001) as compared with least level of protein. Feed restriction decreased carcass percentage (p<0.001) and increased thigh percentage (p<0.01) at 32 day of age. Carcass percentage (P<0.05) and breast percentage increased in broilers fed on low-energy diets compared with those fed on high-energy diets during different periods. The low protein diet decreased carcass percentage (p<0.01), breast percentage (p<0.001) during different periods and thigh percentage (p<0.05) at 21 day of age. The result of this experiment indicated that the lowest protein level had the highest growth hormone concentration at 49 day of age. The low energy diet increased growth hormone concentration (p<0.05) at 21 day of age.
 
 
 
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