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Articles by R. Puttha
Total Records ( 2 ) for R. Puttha
  R. Puttha , S. Jogloy , S. Wongkaew , J. Sanitchon , T. Kesmala and A. Patanothai
  The objectives of this study were to estimate heritability for PBNV resistance parameters and to determine phenotypic and genotypic correlations among PBNV resistance parameters and agronomic traits. One hundred and ninety two progenies in the F5 and F6 generations of peanut as well as their parents were evaluated under natural infection of PBNV in a randomized complete block design with six replications for two years. Additional experiment of the duplicated materials was also conducted for agronomic evaluation for one year using similar experimental procedures under conditions that favored optimum agronomic performance. Heritability estimates were low to relatively high for both disease incidence and disease severity, depending on crosses, ranging form 0.10 to 0.90 and 0.00 to 0.79, respectively. Phenotypic and genotypic correlation coefficients for PBND incidence and PBND severity were high, ranging from 0.94** to 0.98** and 0.99** and 1.00**, respectively. Phenotypic and genotypic correlation coefficients between PBNV resistance parameters and agronomic characters were generally low for most pairs of characters, except between PBNV resistance parameters and 100-seed weight for which it was moderate (0.42** to 0.60**) PBNV susceptibility is somewhat associated with larger seed and might hinder the progress of breeding for large-seeded peanut with resistance to PBNV.
  S. Sun , R. Puttha , S. Ghezaiel , M. Skae , C. Cooper and R. Amin
  Objective: To determine the effect of coeliac disease and treatment with a gluten-free diet on growth and glycaemic control in asymptomatic children with Type 1 diabetes.
Methods: Data were compared in children with coeliac disease diagnosed by annual antibody screening and jejunal biopsy and treated with a gluten-free diet (n = 49) against individuals who were antibody negative (n = 49) matched for age, sex and duration of diabetes.
Results: No differences in growth were observed. In the years prior to diagnosis of coeliac disease, mean glycated haemoglobin (HbA1c) was lower in cases compared with control subjects [8.3 ± 1.1% vs. 8.7 ± 0.9%, P = 0.02 (mean ± sd)]. In cases, HbA1c deteriorated 12 months from the start of a gluten-free diet to levels similar to control subjects (8.9 ± 1.5% vs. 8.8 ± 1.5%, P-value for analysis of variance = 0.9). In regression analysis, the diagnosis of coeliac disease and start of a gluten-free diet was associated with a rise in HbA1c in the first year of treatment [odds ratio 1.56 (95% confidence intervals 1.16–2.10), P = 0.003] after adjusting for insulin dose and regimen and other variables.
Conclusions: In children with Type 1 diabetes, lower HbA1c prior to diagnosis of silent coeliac disease rises following treatment with a gluten-free diet to levels similar to those without coeliac disease. Although unproven, these observations may relate to abnormalities at the small bowel mucosa before the appearance of circulating coeliac antibodies.
 
 
 
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