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Articles by Q Qi
Total Records ( 2 ) for Q Qi
  J Liao , Q Qi , X Zhu , Y Cao and T. Li
 

IP multimedia subsystem (IMS) is over IP network architecture, but mobile IP cannot directly support session mobility controlled by session initiation protocol-based signaling. The long signaling delay for session reestablishment in application layer always results in session interruptions during the handoff. Therefore, handoff poses a challenge for quality of service (QoS) maintenance in IMS that targets to offer real-time multimedia applications over wireless mobile networks. The existing approaches to solve this problem depend on the advance resource reservation and the optimization of handoff control. Unfortunately, big cost of the advance resource reservation in neighboring domains is a major problem that leads to a serious signaling load and a waste of wireless bandwidth. To solve this issue, we present an enhanced IMS handoff mechanism (EHM) based on user mobility prediction to save network resources by avoiding multiple useless advance reservations. In addition, to support the heterogeneous access networks in IMS domain, EHM evolves a network selective scheme to utilize the network resources more efficiently. The architecture of EHM and the advance QoS negotiation signaling are also presented. We model the cost, the handoff delay and the session blocking probability for EHM and the previous work. Analytical and simulation results show that EHM can enhance the handoff performance, such as reducing resource reservation cost greatly, decreasing session reestablishment delay and making good use of multiple access network resources.

  Y Wu , H Li , R. J. F Loos , Q Qi , F. B Hu , Y Liu and X. Lin
 

We previously found that plasma RBP4 levels were strongly associated with metabolic syndrome components. This study aimed to determine whether RBP4 variants are associated with the metabolic syndrome components and plasma RBP4 levels, and to investigate whether the associations between plasma RBP4 and the metabolic syndrome components are causal. Five tagSNPs were tested for their associations with plasma RBP4 levels and metabolic syndrome components in a population-based sample of 3,210 Chinese Hans. A possible causal relationship between plasma RBP4 levels and hypertriglyceridemia was explored by Mendelian randomization. Plasma RBP4 levels were significantly associated with rs10882273 (βz –0.10SD[–0.17, –0.03], P = 0.0050), rs3758538 (βz –0.13SD[–0.24, –0.02], P = 0.0249) in all participants, and with rs17108993 in Shanghai participants (βz –0.19SD[–0.32, –0.05], P = 0.0061). The single nucleotide polymorphism (SNP) rs3758538 was significantly associated with hypertriglyceridemia (OR 0.62[0.45–0.85], P = 0.0026) and triglycerides (βz –0.19SD[–0.30, –0.07], P = 0.001) in all participants. In Mendelian randomization analysis, the observed effect size of association between rs3758538 and hypertriglyceridemia was different from the expected effect size (P = 0.0213). This is the first study to show that the RBP4 variants are significantly associated with plasma RBP4 levels and hypertriglyceridemia risk in Chinese Hans. However, results of Mendelian randomization do not support the hypothesis that RBP4 levels are causally related to hypertriglyceridemia risk.

 
 
 
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