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Articles by N. Wang
Total Records ( 3 ) for N. Wang
  K. Xiao , X. Zhu , K.M. Peng , N. Wang and H. Song
  The purpose of this study was to elucidate the function of visfatin in the mucosal immune barrier of weaned piglets by observing histological changes in the small intestine and visfatin expression in LPS-induced piglets. The mucosal structure of the small intestine was damaged. The duodenum, jejunum, ileum and the intestinal villous were atrophied and shorter. Epithelial lymphocytes and lamina propria lymphoid tissue was reduced and aggregates of lymphoid nodules were increased. Visfatin-positive products were expressed in the small intestinal mucosa and mainly distributed in lamina propria lymphoid tissue. After LPS administration, visfatin-positive cells were reduced in the epithelial lymphocytes, lamina propria and intestinal gland but increased in aggregates lymphoids. This study suggested that an LPS-induced immune stress model could induce structural changes of piglet small intestine and cause changes of intestinal mucosal immune barrier function. In addition, the distribution and quantity of visfatin-positive cells were altered, suggesting visfatin is involved in the humoral immune responses of piglets.
  D.S. Achor , E. Etxeberria , N. Wang , S.Y. Folimonova , K.R. Chung and L.G. Albrigo
  This study was undertaken to develop a better understanding of the relationship between symptom development and the nature of the citrus disease, huanglongbing. The most characteristic symptom of huanglongbing (HLB) is the non-symmetrical mottled chlorosis of leaf blades. Starch accumulation and phloem collapse have been associated with symptom development in this disease presumed to be caused by Candidatus Liberibacter asiaticus. Several hypotheses regarding phloem disruption to starch accumulation to chlorosis evolved concerning symptom development. These were tested using light and Transmission Electron Microscopy (TEM). Samples collected and fixed for TEM with various stages of HLB symptoms, revealed the following: starch accumulation occurred after phloem plugging and cell collapse and therefore, localized carbohydrate deficiency may be a factor. Starch packing of chloroplasts did not rupture the outer membranes, but the inner grana structure was disrupted thus, leading to chlorosis. This occurred only in parts of the leaf where phloem plugging occurred. Sieve elements were obstructed by both amorphous and filamentous materials and both occurred in readily observed amounts, while bacteria were insufficient to directly cause plugging. The amorphous material was positively identified as callose by immunoassay with gold labeling. Phloem protein 2 was identified in the filamentous plugging material using immunoassay with gold labeling. This information supports the development of HLB symptoms in the following sequence: phloem plugging and necrosis with cell wall swelling of sieve elements and companion cells followed by some phloem cell collapse, presumed sugar backup in localized leaf blade areas leading to starch accumulation until chloroplast structure is disrupted with resulting chlorosis.
  H Chen , J Miao and N. Wang
 

We develop a dynamic incomplete-markets model of entrepreneurial firms, and demonstrate the implications of nondiversifiable risks for entrepreneurs' interdependent consumption, portfolio allocation, financing, investment, and business exit decisions. We characterize the optimal capital structure via a generalized tradeoff model where risky debt provides significant diversification benefits. Nondiversifiable risks have several important implications: More risk-averse entrepreneurs default earlier, but choose higher leverage; lack of diversification causes entrepreneurial firms to underinvest relative to public firms, and risky debt partially alleviates this problem; and entrepreneurial risk aversion can overturn the risk-shifting incentives induced by risky debt. We also analytically characterize the idiosyncratic risk premium.

 
 
 
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