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Articles by Jianhong Yao
Total Records ( 3 ) for Jianhong Yao
  Jicheng Liu , Jianhong Yao and Xia Liu
  A method used to optimize the width of the window function by measuring energy concentration of S transform is discussed in the study which is called adaptive optimized generalized S-transform (AOGST). Adaptive parameter which can adjust the width of window function adaptively in frequency domain according to the different component in signal and achieve the optimized time-frequency distribution is induced in S transform. The optimization scheme has been proposed and a set of test signals have been evaluated. The results indicated that the method can provide much improved energy concentration in time-frequency domain, compared with standard S transform, short Fourier transform and its adaptive forms. Finally, the method has been used to analyze the time-frequency distribution of seismic signal and demonstrated its effectiveness.
  Jianhong Yao , Lingyu Zhang , Daxing Sun and Jianbai Song
  As the disadvantages of the conventional ant colony optimization, such as convergence speed and local optimal value, an improved multi-agent ant colony optimization for reactive power optimization method is proposed. The improved sort-weighted ant colony optimization is combined with multi-agent system to improve convergence speed and calculation accuracy and to avoid falling into local optimal value effectively. Improved algorithm is applied to reactive power optimization in IEEE 30-node systems, the simulation results verify the validity of the algorithm.
  Jerzy Frczek , Tae Whan Kim , Hui Xiao , Jianhong Yao , Qian Wen , Yali Li , Jean-Laurent Casanova , Juliusz Pryjma and Xiaoxia Li
  Two parallel interleukin-1 (IL-1)-mediated signaling pathways have been uncovered for IL-1R-TLR-mediated NFκB activation: TAK1-dependent and MEKK3-dependent pathways, respectively. The TAK1-dependent pathway leads to IKKα/β phosphorylation and IKKβ activation, resulting in classic NFκB activation through IκBα phosphorylation and degradation. The TAK1-independent MEKK3-dependent pathway involves IKKγ phosphorylation and IKKα activation, resulting in NFκB activation through dissociation of phosphorylated IκBα from NFκB without IκBα degradation. IL-1 receptor-associated kinase 4 (IRAK4) belongs to the IRAK family of proteins and plays a critical role in IL-1R/TLR-mediated signaling. IRAK4 kinase-inactive mutant failed to mediate the IL-1R-TLR-induced TAK1-dependent NFκB activation pathway, but mediated IL-1-induced TAK1-independent NFκB activation and retained the ability to activate substantial gene expression, indicating a structural role of IRAK4 in mediating this alternative NFκB activation pathway. Deletion analysis of IRAK4 indicates the essential structural role of the IRAK4 death domain in receptor proximal signaling for mediating IL-1R-TLR-induced NFκB activation.
 
 
 
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