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Articles by H.M. Tukur
Total Records ( 2 ) for H.M. Tukur
  A. Abubakar , H.M. Tukur , A.A. Sekoni and W.A. Hassan
  A total of 120 birds were divided into 5 groups of twenty four birds each. Each group was regarded as treatment. The treatment was subdivided into 3 groups of 8 birds each as replicates. Treatment 1 served as control diet with no rice bran and yeast supplementation, diet 2 and 3 contained 30 and 35% rice bran without yeast supplementation, respectively. Diets 4 and 5 were the same as diets 1 and 2 except that 2 g kg-1 of yeast was added to each. Between 45th and 50th week of age of the birds egg quality trial was conducted to assess haugh unit, shell thickness and yolk index. Results of feed intake, hen day egg production, feed conversion ratio and mortality were not significantly (p<0.05) influenced by dietary treatments. Haugh unit, yolk index and average weight of egg did not vary significantly (p<0.05) between treatments, however, shell weight and shell thickness were significantly affected by the treatments. Total cost of production was higher (p<0.05) for the control group compared to treatments 2, 3 and 5. Revenue generated from sale of eggs was higher for birds on treatment 4 compared to those on treatments 1 and 5 (p<0.05). Net farm income was higher for treatments 2, 3 and 4 compared to the control. It could be concluded from the results of this study that performance of birds was not affected by the levels of rice bran with and without yeast supplementation. However, shell thickness, shell weight and net farm income were significantly affected by the treatments.
  E.A. Ogunsan , A.A. Seawright , D.O. Ehizibolo , H.M. Tukur , M.S. Ahmed , Y.G. Dashe and M.Y. Sugun
  It has become apparent that inflammation provoked by injury to renal epithelial cells serves to amplify kidney injury and dysfunction in vivo Compounds including the sesquiterpene ketones generally believed to cause kidney injury by direct tubular toxicity or crystal nephropathy have been considered in different renal studies. The aim of this study was to highlight the histotoxicity of Deisopropylngaione (DIN) in mice. Ninety male quackenbush mice weighing between 30 and 35 g b.wt. were divided into five groups of fifteen mice per group and received 70, 98, 137, 192 and 268 mg kg-1 DIN intraperitoneally, respectively. Haematoxylin erosin (H and E) and Von Kossa stained histology slides showed proximal tubular necrosis and hyaline cast formation. Deposition of calcium salt in residual necrotic tubular epithelium was on the increase with increase in dose rate. Besides the pulmonary lesions seen in mice following intoxication by DIN, the kidney lesions were consistent with previous studies of these compounds in mice and similar to mercuric chloride poisoning in sheep.
 
 
 
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