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Articles by Fei Shi
Total Records ( 2 ) for Fei Shi
  Ling Zhao , Kaiyu Wang , Ruqi Yang , Xun Wang , Defang Chen and Fei Shi
  To deeply explore the pharmacological activities of the Chinese truffle Tuber sinense (CT) in this study, three kinds of the crude polysaccharides from Chinese Truffle Tuber sinense (CTCP) were extracted by the method of water extracting-alcohol precipitating which include Water-soluble Polysaccharide (W-CTCP) and Alkali-soluble Polysaccharides (A-CTCPI, A-CTCPII) and then the polysaccharide content of the CTCP were determined. The antioxidant activities of the CTCPs were evaluated by reducing power, superoxide radical and hydroxyl radical free radical-scavenging assay. As the results, the polysaccharide (W-CTCP, A-CTCPI and A-CTCPII) content was 46.76, 74.05 and 57.68%, respectively. The polysaccharides all exhibited antioxidant activities in vitro and W-CTCP showed the strongest activity. It can be concluded that the polysaccharides extracted from CT should be explored as a potential natural antioxidant agent for use in functional foods or medicine.
  Fei Shi , Xiaolan Seng , Huaqiao Tang , Shiming Zhao , Yang Deng , Ruwen Jin and Yinglun Li
  This study was conducted to investigate the effects of feeding corns naturally contaminated with AflatoxinB1 (AFB1) on performance, serum biochemistry, liver histopathological changes and percentage of hepatocyte apoptosis of Cherry Valley Ducks. Sixty, 1 day old ducks randomly divide into groups and were fed corn-soybean meal uncontaminated or contaminated AFB1. AFB1-contaminated diet significantly decreased the Body Weight (BW) gain, feed intake and feed conversion rate (p<0.05). The concentration of serum protein and the activity antioxidant enzyme were significantly decreased (p<0.05), the activity of serum biochemical enzyme and hepatocyte apoptosis were significantly increased (p<0.05) in AFB1-contaminated group. From histological study, liver tissue of ducks receiving AFB1-contaminated diets had markedly bile duct hyperplasia and vacuolar degeneration.
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