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Articles by Burhanuddin Bahar
Total Records ( 2 ) for Burhanuddin Bahar
  Ressy Dwiyanti , Mochammad Hatta , Rosdiana Natzir , Sutji Pratiwi , Muhammad Sabir , Yadi Yasir , Rizki Amelia Noviyanthi , Ade Rifka Junita , Nataniel Tandirogang , Masyhudi Amir , Matius Fias , Juhri Saning and Burhanuddin Bahar
  Only small proportions of Salmonella enterica serovar Typhi (S. Typhi), in endemic areas, exposing individuals develop typhoid fever symptoms. Identification of polymorphisms NOD2, VDR and NRAMP1 genes is mandatory for a better understanding of typhoid fever molecular pathogenesis and thus to the development of novel strategies for the prevention of infection. The aim of this study was to determine whether genetic polymorphisms in nucleotide oligomerization binding domain 2 (NOD2), vitamin D receptor (VDR) gene and natural resistance-associated macrophage protein 1 (NRAMP1) genes are involved in host susceptibility to severity of typhoid fever. The genotyping of eight regions were applied in the genes; NOD2, VDR and NRAMP1, using PCR-RFLP. A multivariate analysis on 426 mild and 35 severe state of typhoid fever patients. All patients living in the geographically isolated village of South Sulawesi, Middle Sulawesi, Southeast Sulawesi, East Borneo and Papua islands which was an endemic areas in Indonesia. Data were analyzed using Microsoft Excel and Stata 9.2. The G/C and C/C alleles of exon 8 in NOD2 gene were strongly associated and more frequently found in the patients with severe typhoid fever than in mild typhoid fever (p= 0.027and 0.014, odds ratio = 16.7and 27.9, 95% confidence interval = 3.4 - 25.7and 2.6 - 37.1in G/C allele and C/C allele), respectively. In contrast, No evidence for the association of VDR and NRAMP1 genes polymorphisms with severity state of typhoid fever. The polymorphism of exon 8 in NOD2 gene; heterozygotes or homozygotes for a G→C change in codon 2722, was related to susceptibility to typhoid fever in clinical severity of this disease. Polymorphisms of exon 8 in NOD2 gene; heterozygotes or homozygotes for G/C in codon 2722 were related to susceptibility to typhoid fever in clinical severity.
  R. Erni Yetti , Muhammad Syafar , Andi Zulkifli , Rahayu Indriasari , Burhanuddin Bahar , Suriah , Agus Bintara Birawida , Bagoes Widjanarko , Zadrak Tombeg , Saskiyanto Manggabarani and Anto J. Hadi
  Background and Objective: Obesity is a major nutritional problem among adolescences and is becoming an epidemic. Globally, it is estimated that obesity is the fifth main cause of death and many factors underlie this problem. The rise in obesity prevalence in children and adolescents is due to environmental factors, such as diet and physical activity. This study aimed to examine a family-based empowerment model to prevent obesity in adolescents in Tana Toraja. Materials and Methods: This was a quasi-experimental study that was conducted using pre- and post-test measurements with a control group design. A total of 64 study participants were divided into two groups: 32 adolescents in the intervention group and 32 others in the control group. Data were obtained through a structured questionnaire regarding nutritional knowledge, attitude and behaviour. Nutritional status was determined after measuring weight and height by using digital scales. In addition, eating behaviours were measured using a food frequency questionnaire and 2 24 h recall forms. Results: The results showed that there were differences in nutritional knowledge (p< 0.001), nutritional attitudes (p<0.001) and nutritional behaviour (p<0.001) in both the intervention and control groups after the implementation of the family-based empowerment programme. The results of this study also showed that a family-based empowerment model for obesity prevention can be applied in the family and school settings in Indonesia. Conclusion: Nutritional education through the empowerment of families regarding the prevention of obesity has positive impacts on nutrition knowledge, attitudes and behaviour of adolescents, providing the tools to adopt an effective and controlled diet.
 
 
 
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