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Articles by Bei Liu
Total Records ( 2 ) for Bei Liu
  Han Renting , Xuan Wang , Nan Geng , Wenhao Suo , Bei Liu and Yuxiang Huang
  This study utilizes the Cobb-douglas production function and regression analysis to establish a computing model for the contribution rate of agricultural mechanization to agricultural production. The agricultural mechanization and agricultural output data of the Shaanxi Province from 2001 to 2010 were used as examples for the estimation. The results demonstrate that the contribution of agricultural mechanization to agricultural output was 31.46%. A regression analysis of the various input factors revealed that the growth of agricultural output was primarily due to an increase in capital and agricultural machinery. Capital investment displayed the largest contribution to the output, followed, in descending order, by the investment in agricultural machinery and land investment; moreover, the labor investment did not significantly affect the output. With an increased agricultural machinery and capital investment, the agricultural output of the Shaanxi Province exhibited a trend of significant upward growth. This observation suggests that further investment in agricultural machinery and an increase in the mechanization level of agricultural production may extensively contribute to the growth of the agricultural output. The results of this study are relevant for future evaluations of the development level of agricultural mechanization and its potential contribution to the agricultural output.
  Li-xia Yang , Rui-wei Guo , Bei Liu , Xian-mei Wang , Feng Qi , Chuan-ming Guo , Yan-kun Shia and Hong Wang
 

Endothelial dysfunction is associated with cardiovascular diseases. The Ca2+ influx occurring via activation of plasmalemma Ca2+ channels was shown to be critical in signaling the increase in endothelial permeability in response to a variety of permeability-increasing mediators. It has been reported that angiotensin II (AngII) could induce Ca2+ signaling in some cells, and transient receptor potential canonical 1 (TRPC1) had an important role in this process. The objective of this study was to examine the mechanism of AngII-induced Ca2+ entry and vascular endothelial hyperpermeability. Human umbilical vein endothelial cells (HUVECs) exposed to AngII exhibited dose-dependent increase in [Ca2+]i and endothelial permeability. Quantitative real-time RT-PCR and Western blotting showed that the level of TRPC1 expression had increased significantly at 12 h and at 24 h after treatment of HUEVCs with AngII. The expression of p65 was suppressed using an RNAi strategy. The results showed that the NF-κB signaling pathway and type-1 receptor of AngII was involved in AngII-induced TRPC1 upregulation. Moreover, knockdown of TRPC1 and NF-κB expression attenuates AngII-induced [Ca2+]i and endothelial permeability. NF-κB and TRPC1 have critical roles in AngII-induced Ca2+ entry and endothelial permeability.

 
 
 
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