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Articles by B Jin
Total Records ( 3 ) for B Jin
  O Itano , K Yang , K Fan , N Kurihara , H Shinozaki , S Abe , B Jin , C Gravaghi , W Edelmann , L Augenlicht , L Kopelovich , R Kucherlapati , S Lamprecht and M. Lipkin

We have previously reported that sulindac, a non-steroidal anti-inflammatory drug, inhibited tumor formation in the small intestine but increased tumors in the colon of ApcMin/+ mice, a model of human familial adenomatous polyposis. To further explore intestinal regional responses, we studied effects of sulindac on additional gene-targeted mouse models of human intestinal tumorigenesis; these were (i) Apc1638N/+ mouse (chain termination mutation in exon 15 of the Apc gene); (ii) Mlh1+/– mouse (DNA mismatch repair deficiency, a mouse model of human hereditary non-polyposis colorectal cancer) and (iii) double-heterozygous Mlh1+/–Apc1638N/+ mutant mouse. Mice were fed AIN-76A control diet with or without 0.02% sulindac for 6 months. Intestinal regional tumor incidence, multiplicity, volume and degree of inflammation were used as end points. The results showed the following: (i) sulindac inhibited tumor development in the small intestine of Apc1638N/+ mice; (ii) in contrast, sulindac increased tumors in the small intestine of Mlh1 mutant mice, a neoplastic effect which persisted in heterozygous compound Mlh1+/–Apc1638N/+ mutant mice; (iii) sulindac increased tumors in the cecum of all mice regardless of genetic background; (iv) sulindac decreased inflammation in the small intestine of Apc1638N/+ mice, but it increased inflammation in the small intestine of Mlh1+/ mice and Mlh1+/–Apc1638N/+ mice and (v) sulindac enhanced inflammation in the cecum of all mutant mice. Findings indicate that the effects of sulindac in the intestine of these mutant mouse models are probably related to genetic background and appear to be associated with its inflammatory-inducing response.

  B Jin , X Zhao , Z Long , F Qi and S. Yu

The adoption of radio frequency identification (RFID) tags and devices facilitates the observation and monitoring of event patterns, while highly temporal restrained RFID data further make events complicated. Although the existing Pub/Sub systems can help distributed applications monitor and disseminate interesting events, they are unable to express RFID-related events directly. Extending a system for new RFID requirements not only needs a lot of work, but also provides no performance guarantees. It is, therefore, necessary to design and implement an effective and efficient Pub/Sub system in order to capture and disseminate RFID-related events. This paper presents a composite subscription specification for RFID-related application scenarios. It enables the subscription of predicates for RFID code as well as various complex events. Moreover, this paper proposes the algorithms for RFID-code subscription matching and complex events detection which have been implemented in our Pub/Sub system, OncePubSub. Also, experiments were conducted to quantify the performance and overhead of the above algorithms. Performance evaluation results indicate that OncePubSub is more efficient than SIENA- and JESS-based systems.

  B Jin and J. Zou

A finite-element method is proposed for the nonlinear inverse problem of estimating the Robin coefficient in a stationary diffusion equation from boundary measurements of the solution and the heat flux. The inverse problem is formulated as an output least squares optimization problem with an appropriate regularization, then the finite-element method is employed to discretize the nonlinear optimization system. Mathematical properties of both the continuous and the discrete optimization problems are investigated. The conjugate gradient method is employed to solve the optimization problem, and an efficient preconditioner via the Sobolev inner product is also suggested. Numerical results for several two-dimensional problems are presented to illustrate the efficiency of the proposed algorithm.

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