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Articles by A.S. Fairchild
Total Records ( 2 ) for A.S. Fairchild
  A.S. Fairchild , J.L. Grimes , J.K. Porter , W.J. Croom Jr. , L.R. Daniel and W.M. Hagler Jr.
  Turkey poults were randomly placed in batteries and fed one of four dietary treatments: control (C); control plus 4ppm diacetoxyscirpenol (DAS); control plus 300 ppm (FA); and control plus 4ppm DAS and 300ppm FA (FD). There were 10 poults per pen with 6 replicate pens per treatment. Individual BW, BW gains (BWG) and feed consumption by pen was determined at d6, d12, and d18. Period and cumulative feed to gain was calculated. Mouth lesions were scored for treatments at d18. On d18 poults were euthanized for determination of organ weights and jejunal histomorphometrics. FA had no effect on BW or BWG at any period compared to C. Poults fed FD had reduced BW and BWG compared to C, while poults fed DAS had lower BW than all treatments at every period. Poults fed FA or C had better feed to gain (P<0.05) than poults fed DAS or FD at d6. There were no differences among the treatments at d12 or d18. Poults fed FA had significantly lower relative intestine wt than poults fed other diets, and significantly higher relative bursa wt at d18 when compared to poults fed DAS or FD. DAS, FA and FD altered intestinal architecture. Poults fed DAS or FD had higher mouth lesion scores than poults fed FA or C, but mouth lesion scores in DAS and FD poults were not different from each other. Dietary DAS resulted in decreased poult performance, while dietary FA had little or no effect. Fusaric acid fed in combination with DAS resulted in some protective effect towards DAS.
  A.S. Fairchild , J. Croom , J.L. Grimes and W.M. Hagler, Jr.
  The objectives of this study were: 1) to examine effects of a dietary sodium bentonite, ASTRA-BEN-20® (AB20) [Prince Agri Products, Inc., Quincy, Ill.], on in vitro binding of T2 toxin and aflatoxin B1 (AFB1); and 2) to examine the effects of AB20 in broiler chicks fed 6 ppm dietary T2 toxin (T2) or 4 ppm aflatoxin B1 (AFB1) to 21 d. Day-old male broiler chicks were randomly placed in batteries and assigned to one of nine dietary treatments (7 replicates of 5 chicks): control (C); 1% AB20; 2% AB20; T2; T2+ 1% AB20; T2+2% AB20; AFB1; AFB1+1% AB20; and AFB1+2% AB20. Individual BW, feed consumption by pen, period and cumulative feed conversions (PFC and CFC, respectively), and BW gains (BWG) were determined weekly. Liver weight (LW), relative liver weight (RLW), and liver lipid (%, PLL) were determined at d21 for AFB1 chicks. Oral lesions were scored on d21 for T2 chicks. One and 2% AB20 reduced BW during wk 1 and 2 compared to controls. AFB1 reduced BW during wk 2 and 3 and increased PLL at d21. Both 1 and 2% AB20 returned BW, BWG, and PLL to control levels. T2 reduced BW and BWG at wk 1, 2, and 3. T2 + 1% AB20 and T2 + 2% AB20 improved BW at wk 1 and 2 and BWG at wk 1 while T2 + 1% AB20 improved BW at wk 3 when compared to T2. T2 + 2% AB20 also decreased oral lesion severity compared to T2 at d21. PFC and CFC were increased by T2 and T2 + 1% AB20. CFC tended to improve when chicks were fed T2 + 2% AB20 (P<0.06). AB20 protected broiler chicks from dietary AFB1 and provided some protection against dietary T2.
 
 
 
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