Intracranial hypertension is initially an alarm signal for the increased ICP; later the increase in intracranial pressure is accompanied by symptoms and there is the ICH syndrome; later on the increase in ICP becomes a pathogenic mechanism in itself and intracranial hypertension appears as an acute disease. The pattern of ICH includes a three-phase evolution; the pressure-time fluctuation is the dynamic element in the progression and decompensation of intracranial hypertension. The evolution of ICH is made by exceeding the critical thresholds of the ICP equivalent to each stage and the decompensation corresponds to the acute critical pressure-time fluctuation. The main features that determine clinical course of intracranial hypertension are: the speed of ICP increase till and over the normal limit, the critical thresholds and the highest value of ICP, the period of pathologic value of ICP, the length of recurrence to normal value and the frequent occurrence of ICP increase. There are five main forms of ICP increase depending of the rapidity of ICP increase till normal limit and over. Intracranial hypertension is classified in four forms based on the etiopathogenesis: parenchymatous intracranial hypertension with an intrinsic cerebral cause, vascular intracranial hypertension, which has its etiology in disorders of the cerebral blood circulation, meningeal intracranial hypertension and idiopathic intracranial hypertension, the former pseudotumor cerebri; it is an incomplete ICH syndrome. PDFFulltextXMLReferencesCitation
How to cite this article
St. M. Iencean, 2004. Pattern of Increased Intracranial Pressure and Classification of Intracranial Hypertension. Journal of Medical Sciences, 4: 52-58.